RC Holley

First name:
RC
Last name:
Holley
Smith, K. J., Self, R. L., Butler, T. R., Mullins, M. M., Ghayoumi, L., Holley, R. C., et al. (2007). Methamphetamine exposure antagonizes N-methyl-D-aspartate receptor-mediated neurotoxicity in organotypic hippocampal slice cultures. Brain Research, 1157, 74-80. https://doi.org/10.1016/j.brainres.2007.04.056 (Original work published 2007)
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LH, W., Prendergast, M. A., Blanchard, J., Holley, R. C., Chambers, E. R., & Littleton, J. M. (2006). Potential value of changes in cell markers in organotypic hippocampal cultures associated with chronic EtOH exposure and withdrawal: comparison with NMDA-induced changes. Alcoholism, Clinical And Experimental Research, 30(10), 1768-80. https://doi.org/10.1111/j.1530-0277.2006.00210.x
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Prendergast, M. A., Harris, B. R., Mullholland, P. J., JA, 2nd B., Gibson, D. A., Holley, R. C., & Littleton, J. M. Hippocampal CA1 region neurodegeneration produced by ethanol withdrawal requires activation of intrinsic polysynaptic hippocampal pathways and function of N-methyl-D-aspartate receptors. Neuroscience, 124(4), 869-77. https://doi.org/10.1016/j.neuroscience.2003.12.013 (Original work published 2004)
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Harris, B. R., Gibson, D. A., Prendergast, M. A., Blanchard, J. A., Holley, R. C., Scotland, R. L., et al. (2003). The neurotoxicity induced by ethanol withdrawal in mature organotypic hippocampal slices might involve cross-talk between metabotropic glutamate type 5 receptors and N-methyl-D-aspartate receptors. Alcoholism, Clinical And Experimental Research, 27(11), 1724-35. https://doi.org/10.1097/01.ALC.0000093601.33119.E3
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Mulholland, P. J., Harris, B. R., Wilkins, L. H., Self, R. L., Blanchard, J. A., Holley, R. C., et al. (1969). Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression. Alcohol (Fayetteville, N.y.), 31(1-2), 1-10. https://doi.org/10.1016/j.alcohol.2003.09.001 (Original work published 1969)
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Gibson, D. A., Harris, B. R., Prendergast, M. A., JA, 2nd B., Holley, R. C., Pedigo, N. W., et al. (2003). Polyamines contribute to ethanol withdrawal-induced neurotoxicity in rat hippocampal slice cultures through interactions with the NMDA receptor. Alcoholism, Clinical And Experimental Research, 27(7), 1099-106. https://doi.org/10.1097/01.ALC.0000075824.10502.DD
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Harris, B. R., Prendergast, M. A., Gibson, D. A., Rogers, D. T., Blanchard, J. A., Holley, R. C., et al. (2002). Acamprosate inhibits the binding and neurotoxic effects of trans-ACPD, suggesting a novel site of action at metabotropic glutamate receptors. Alcoholism, Clinical And Experimental Research, 26(12), 1779-93. https://doi.org/10.1097/01.ALC.0000042011.99580.98
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Mayer, S., Harris, B. R., Gibson, D. A., Blanchard, J. A., Prendergast, M. A., Holley, R. C., & Littleton, J. (2002). Acamprosate, MK-801, and ifenprodil inhibit neurotoxicity and calcium entry induced by ethanol withdrawal in organotypic slice cultures from neonatal rat hippocampus. Alcoholism, Clinical And Experimental Research, 26(10), 1468-78. https://doi.org/10.1097/01.ALC.0000033261.14548.D2
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Mayer, S., Harris, B., Gibson, D. A., Blanchard, J., Prendergast, M. A., Holley, R. C., & Littleton, J. (2002). Acamprosate has no effect on NMDA-induced toxicity but reduces toxicity induced by spermidine or by changing the medium in organotypic hippocampal slice cultures from rat. Alcoholism, Clinical And Experimental Research, 26(5), 655-62.
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Prendergast, M. A., Harris, B. R., Mayer, S., Holley, R. C., & Littleton, J. M. (1969). Nicotine exposure reduces N-methyl-D-aspartate toxicity in the hippocampus: relation to distribution of the alpha7 nicotinic acetylcholine receptor subunit. Medical Science Monitor : International Medical Journal Of Experimental And Clinical Research, 7(6), 1153-60. (Original work published 1969)
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