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RL Self

First name:
RL
Last name:
Self
Mulholland, P. J., Stepanyan, T. D., Self, R. L., Hensley, A. K., Harris, B. R., Kowalski, A., et al. Corticosterone and dexamethasone potentiate cytotoxicity associated with oxygen-glucose deprivation in organotypic cerebellar slice cultures. Neuroscience, 136(1), 259-67. https://doi.org/10.1016/j.neuroscience.2005.07.043 (Original work published 2005)
Prendergast, M. A., Rogers, D. T., Mulholland, P. J., Littleton, J. M., LH, W., Self, R. L., & Nath, A. (2002). Neurotoxic effects of the human immunodeficiency virus type-1 transcription factor Tat require function of a polyamine sensitive-site on the N-methyl-D-aspartate receptor. Brain Research, 954(2), 300-7. https://doi.org/10.1016/s0006-8993(02)03360-7 (Original work published 2002)
Mulholland, P. J., Harris, B. R., Wilkins, L. H., Self, R. L., Blanchard, J. A., Holley, R. C., et al. (1969). Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression. Alcohol (Fayetteville, N.y.), 31(1-2), 1-10. https://doi.org/10.1016/j.alcohol.2003.09.001 (Original work published 1969)
Self, R. L., Mulholland, P. J., Nath, A., Harris, B. R., & Prendergast, M. A. (2004). The human immunodeficiency virus type-1 transcription factor Tat produces elevations in intracellular Ca2+ that require function of an N-methyl-D-aspartate receptor polyamine-sensitive site. Brain Research, 995(1), 39-45. https://doi.org/10.1016/j.brainres.2003.09.052 (Original work published 2004)
Mulholland, P. J., Self, R. L., Harris, B. R., Littleton, J. M., & Prendergast, M. A. (-)-nicotine ameliorates corticosterone's potentiation of N-methyl-d-aspartate receptor-mediated cornu ammonis 1 toxicity. Neuroscience, 125(3), 671-82. https://doi.org/10.1016/j.neuroscience.2004.02.007 (Original work published 2004)
Mulholland, P. J., Self, R. L., Harris, B. R., Littleton, J. M., & Prendergast, M. A. (2004). Choline exposure reduces potentiation of N-methyl-D-aspartate toxicity by corticosterone in the developing hippocampus. Brain Research. Developmental Brain Research, 153(2), 203-11. https://doi.org/10.1016/j.devbrainres.2004.08.008 (Original work published 2004)
Self, R. L., Mulholland, P. J., Harris, B. R., Nath, A., & Prendergast, M. A. (2004). Cytotoxic effects of exposure to the human immunodeficiency virus type 1 protein Tat in the hippocampus are enhanced by prior ethanol treatment. Alcoholism, Clinical And Experimental Research, 28(12), 1916-24. https://doi.org/10.1097/01.alc.0000148108.93782.05
Mulholland, P. J., Self, R. L., Harris, B. R., Little, H. J., Littleton, J. M., & Prendergast, M. A. (2005). Corticosterone increases damage and cytosolic calcium accumulation associated with ethanol withdrawal in rat hippocampal slice cultures. Alcoholism, Clinical And Experimental Research, 29(5), 871-81. https://doi.org/10.1097/01.alc.0000163509.27577.da
Mulholland, P. J., Self, R. L., Stepanyan, T. D., Little, H. J., Littleton, J. M., & Prendergast, M. A. Thiamine deficiency in the pathogenesis of chronic ethanol-associated cerebellar damage in vitro. Neuroscience, 135(4), 1129-39. https://doi.org/10.1016/j.neuroscience.2005.06.077 (Original work published 2005)
Butler, T. R., Smith, K. J., Self, R. L., Braden, B. B., & Prendergast, M. A. (2011). Neurodegenerative effects of recombinant HIV-1 Tat(1-86) are associated with inhibition of microtubule formation and oxidative stress-related reductions in microtubule-associated protein-2(a,b). Neurochemical Research, 36(5), 819-28. https://doi.org/10.1007/s11064-011-0409-2